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Metabolic
scheduleHalf-life: ~7 days (enabling once-weekly dosing)

Survodutide

Survodutide (BI 456906)

Survodutide represents the cutting edge of obesity pharmacology: a dual agonist that activates both GLP-1 and glucagon receptors. While this might seem counterintuitive—glucagon raises blood sugar—the combination produces synergistic effects for weight loss. GLP-1 activation suppresses appetite, slows gastric emptying, and improves insulin secretion, just like semaglutide. Glucagon receptor activation adds something semaglutide lacks: increased energy expenditure through liver-mediated pathways and enhanced fat burning. The result, in phase 2 trials, was up to 19% weight loss at 46 weeks—competitive with tirzepatide's best results. Developed by Boehringer Ingelheim, survodutide is also being studied for NASH (fatty liver disease) and liver fibrosis, where the glucagon component may offer particular benefits by reducing liver fat. The challenge is balancing potency with tolerability; glucagon can raise blood sugar, potentially limiting use in diabetics. As obesity drug development accelerates, survodutide positions itself as a potentially best-in-class option by attacking the problem from multiple angles.

Table of Contents

  • What is Survodutide?
  • Research Benefits
  • How Survodutide Works
  • Research Applications
  • References

What is Survodutide?

Survodutide represents the cutting edge of obesity pharmacology: a dual agonist that activates both GLP-1 and glucagon receptors. While this might seem counterintuitive—glucagon raises blood sugar—the combination produces synergistic effects for weight loss. GLP-1 activation suppresses appetite, slows gastric emptying, and improves insulin secretion, just like semaglutide. Glucagon receptor activation adds something semaglutide lacks: increased energy expenditure through liver-mediated pathways and enhanced fat burning. The result, in phase 2 trials, was up to 19% weight loss at 46 weeks—competitive with tirzepatide's best results. Developed by Boehringer Ingelheim, survodutide is also being studied for NASH (fatty liver disease) and liver fibrosis, where the glucagon component may offer particular benefits by reducing liver fat. The challenge is balancing potency with tolerability; glucagon can raise blood sugar, potentially limiting use in diabetics. As obesity drug development accelerates, survodutide positions itself as a potentially best-in-class option by attacking the problem from multiple angles.

Research Benefits

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Substantial weight loss (up to 19% in trials)

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Appetite suppression via GLP-1

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Increased energy expenditure via glucagon

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May improve fatty liver disease

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Reduces liver fibrosis markers

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Once-weekly dosing

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Potential metabolic benefits beyond weight

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Additive effects from dual mechanism

Research Applications

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Obesity treatment

Active research area with published studies

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Non-alcoholic steatohepatitis (NASH)

Active research area with published studies

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Liver fibrosis

Active research area with published studies

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Metabolic syndrome

Active research area with published studies

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Type 2 diabetes (careful dosing)

Active research area with published studies

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Cardiovascular risk reduction

Active research area with published studies

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Body composition optimization

Active research area with published studies

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Fatty liver disease

Active research area with published studies

Frequently Asked Questions

Scientific References

1

Survodutide: A Dual GLP-1/Glucagon Receptor Agonist for Obesity

The Lancet (2024)

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2

Effects of Survodutide on Body Weight and Liver Fat

New England Journal of Medicine (2024)

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3

Dual Agonism for Metabolic Disease: GLP-1 and Glucagon

Trends in Pharmacological Sciences (2023)

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Quick Reference

Molecular Weight~4,100 Da
Half-Life~7 days (enabling once-weekly dosing)
PurityPharmaceutical grade (clinical trial material)
FormSubcutaneous injection | Pre-filled pen (development)

Sequence

GLP-1/glucagon dual agonist peptide (proprietary modified sequence)

Storage

Refrigerate at 2-8°C | Do not freeze | Protect from light

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